Introduction: Phosphoantigen reactive gamma-delta T-cells, characterised by their expression of a Vdelta2 TCR, have garnered significant interest as immunotherapies against cancers and infectious diseases. These cells can be readily expanded pharmacologically and are capable of mediating a wide variety of immune functions, including pro-inflammatory cytokine production and lysis of target cells. Despite promising results in preclinical studies, the clinical efficacy Vdelta2+ T-cell immunotherapies has been limited. Improving Vdelta2+ T-cell immunotherapies will require treatment protocol refinements to optimise expansion and activation of these cells.
Methods: The aim of this study was to investigate the impact of different antigenic stimuli and routes of administration on Vdelta2+Vgamma9+ T-cell expansion in nonhuman primates. Pigtail macaques were treated with intravenous (IV) or intratracheal (IT) zoledronate, HMB-PP (IV and IT), or IPP (IT), along with IL-2 to stimulate in vivo Vdelta2+Vgamma9+ T-cell expansion. Vdelta2+Vgamma9+ T-cell frequencies and phenotypes were evaluated in blood and tissues sites (lymph nodes, bronchoalveolar lavage fluid [BAL], and rectal mucosa) over time, and compared to baseline samples.
Results: Vdelta2+Vgamma9+ T-cells readily expanded in the peripheral blood of macaques treated with systemic zoledronate or HMB-PP, and in the BAL fluid of macaques treated with IV and IT antigens. Vdelta2+Vgamma9+ T-cell frequencies were consistently low in the lymph nodes and rectal mucosa, with no significant changes in cell frequencies in any of the treatment groups. Expanded Vdelta2+Vgamma9+ T-cells in the blood were predominately CCR6-, which was not observed in Vdelta2+Vgamma9+ T-cells in the BAL.
Conclusion: Vdelta2+Vgamma9+ T-cells readily expand in the blood and airway mucosa of pigtail macaques following treatment with phosphoantigens or bisphosphonates. Systemic phosphoantigen and bisphosphonate mediated expansion with IL-2 preferentially expands CCR6-Vdelta2+Vgamma9+ T-cells in the blood. This work has implications for improving Vdelta2+ T-cell immunotherapies against both infectious and neoplastic diseases.